Anyone who spends years working in pharmacies or clinics comes to appreciate medicines that reinvent how we treat chronic conditions—Fluocinolone Acetonide is one of these turning points. Back in the 1950s and ’60s, scientists sought ways to ease inflammatory skin diseases with fewer side effects than older steroids. The introduction of fluorination in steroid molecules gave a new punch to topical therapies. Adding an acetonide group to triamcinolone opened the door to better skin penetration, meaningful symptom relief, and reduced systemic risk. Fluocinolone Acetonide started as a promise in chemical synthesis labs. Clinical practice soon confirmed its practical value, turning this compound from a novelty into a staple on dermatology shelves worldwide by the late 1960s. Today, it remains a trusted fallback for patients whose suffering demands dependable, predictable relief.
On a typical pharmacy shelf, Fluocinolone Acetonide comes in creams, ointments, lotions, solutions, even ear drops and dental pastes. Each form serves a purpose—creams soak into weepy eczema, ointments suit dry, cracked skin, and scalp solutions work through hair. Many patients walk in with persistent itch, inflamed plaques, and distress, only to find genuine comfort after a few days of regular application. A single active ingredient brings that change. Physicians often reach for this steroid not out of habit but because it delivers results time and again, controlling atopic dermatitis and stubborn eczema where basic moisturizers fall flat.
With a chemical formula of C24H30F2O6 and a molecular weight just over 452 g/mol, Fluocinolone Acetonide stands out for its strong lipophilicity—a property that lets it move smoothly through cell membranes in the upper skin layers. If you’ve ever prepared compounded mixtures, you’ll remember its white crystalline powder dissolves well in acetone, methanol, or chloroform, but hardly in water. Melting points hover around 260°C, which means no ordinary heat can tamper with its structure. The addition of fluorine atoms makes it potent and stable under typical storage conditions. Its specific rotation and UV absorption spectra offer ways for laboratory quality control to verify each batch before it ever reaches a pharmacy.
Any product containing Fluocinolone Acetonide needs precise concentration—usually in the range of 0.01% to 0.025% for topical use. Anything higher risks thinning skin or causing systemic problems if overused. Well-designed labels warn about risks, such as applying on broken skin or long-term use for kids. Batch numbers, shelf life, active and inactive ingredients, and clear directions help steer patients clear of avoidable mistakes. Packaging requirements keep the compound away from light and moisture, preserving both safety and potency until the last squeeze from the tube. This level of care matters for every user, not just the officially “sensitive” ones.
Labs synthesize Fluocinolone Acetonide through a multi-step organic process, beginning with triamcinolone as the backbone. Fluorination follows, which isn’t easy when handling light, volatile reactants. Technicians add the acetonide group using acetone under carefully controlled acidic conditions. Purification demands a watchful eye, with steps like recrystallization and chromatography to weed out byproducts. Making each batch consistently pure involves constant monitoring, precise timing, and attention to even the slightest temperature drift. Finished material faces rigorous chemical and microbiological tests before release. Many experienced compounders say the synthesis mirrors classic examples from steroid chemistry textbooks, marrying old-school chemical craftsmanship with modern safety standards.
Adding fluorine atoms to the steroid nucleus cranks up both the anti-inflammatory power and the ability to resist breakdown by skin enzymes. Chemists over the years have experimented with modifying the side chains or the acetonide group, aiming for steroids that act longer or absorb even better. Certain salt forms increase solubility for newer formulations like gels or sprays. Some labs have explored esterified versions for slow-release implants in ophthalmology or oral gels for mouth lesions. While these changes aim for greater convenience or targeted therapy, the core molecule’s structure drives both its benefits and its toxicity concerns—small tweaks can swing the balance.
Pharmacists, researchers, and patients know Fluocinolone Acetonide under many other names. Look on labels across the world and you’ll spot “Flucort,” “Synalar,” “Fluoderm,” and “Fluonid.” The United States Pharmacopeia tags it as USP Fluocinolone Acetonide. Drug monographs usually flag synonyms like 6α,9α-Difluoro-16α,17α-acetonide-prednisolone, a mouthful but a useful term for scientific communication. Recognizing these alternate names proves essential for anyone needing to cross-reference global literature or check for drug interactions.
Anyone handling medicinal steroids needs solid training. Gloves and eye protection should be non-negotiable in compounding pharmacies and labs. Spills require fast cleanup because even small amounts can sensitize skin over time. Regulatory authorities—like the FDA and EMA—set limits for allowable impurities and require reports on shelf life, stability, and adverse reactions. Quality assurance techs carry out identity, purity, and microbial assays on every production lot, hallmarks of good manufacturing practice. Clinics and hospitals keep strict logs of who used what, when, and on which patient, closing the loop on safety for both staff and users.
Fluocinolone Acetonide creams and lotions treat a vast range of skin issues: eczema, psoriasis, allergic rashes, and stubborn itch from unknown causes. Ear solutions offer relief for chronic otitis externa, helping patients avoid antibiotics. Some eye preparations use micro-doses of this steroid for persistent uveitis or as implants to stop diabetic eye swelling, sparing patients from ongoing injections. Dentists sometimes prescribe pastes with Fluocinolone Acetonide for oral lichen planus—a remarkably tough disease to treat. Its strong anti-inflammatory punch, combined with a reputation for reliability, makes it a daily go-to for doctors working in tough clinical settings.
Teams around the world continue exploring improved delivery systems—microspheres, nanoparticles, foam sprays, and even biodegradable implants—each aiming for better absorption and longer relief with less risk to the skin’s protective barrier. Geneticists and pharmacologists dig deeper into the molecular mechanisms, searching for ways to separate strong anti-inflammatory action from thinning skin and delayed wound healing. Some research groups focus on modeling how long-term use affects local immunity or the skin’s natural microbiome, hoping to inform safer guidelines for chronic conditions. The last decade saw a boom in publications covering new uses, especially for hard-to-treat eye and mouth diseases.
Steroid overuse remains a real-world problem, with never-ending phone calls from worried parents or patients who’ve noticed thinning skin or strange stretch marks. Toxicology research has revealed how repeated or high-dose application can inhibit growth in young patients, trigger adrenal suppression, and set the stage for infections normally ignored by a healthy immune system. Animal studies and large patient series have shown what levels count as safe for adults and kids. Formulators try to minimize risk with lower doses in pediatric versions and special gel bases that deliver less drug per application. Clear guidelines—rotating off steroids, limiting course length, and avoiding face or groin unless urgent—now form the backbone of safety advice.
Drug companies, dermatology groups, and patient advocacy networks keep searching for new answers: How can we deliver this proven molecule with even greater safety? Can one develop smart bandages that release steroid only during flare-ups? What about biodegradable carriers for the ear, eye, or mouth? With gene therapy and biologics drawing attention, some talk as if traditional steroids are on their way out. From years talking with physicians and patients, it’s clear these drugs fill a gap nothing else does, especially in low-resource or fast-relief settings. Expect more research into slow-release technologies, combination creams with antibiotics or antifungals, and tailored advice based on each patient’s lifestyle and genetic profile. The journey that started decades ago has not reached its finish line; Fluocinolone Acetonide has decades of healing and adaptation ahead.
Ask anyone who's lived with eczema, psoriasis, or even relentless itch from bug bites, and you start hearing familiar frustrations. I’ve watched friends rummage through drawers looking for some kind of “miracle cream.” The real-life relief comes from topical steroids, and one of the most trusted among dermatologists is fluocinolone acetonide.
This isn’t the strongest steroid out there, but it packs enough punch for persistent rashes without hammering the skin too hard. Doctors reach for it to calm itching, redness, and swelling. It belongs to the corticosteroid family, medicines known for dialing down the chaos of inflammation.
People facing eczema know that flare-ups don’t just itch—they take over daily life. Fluocinolone acetonide creams and ointments break that cycle. By turning down the immune response locally, swelling and redness ease off. For folks with chronic scalp irritation, shampoos with this ingredient offer a welcome break from embarrassing flakes. Seborrheic dermatitis, a mouthful of a diagnosis, leads to sore, scaly patches around the nose, eyebrows, and hairline for plenty of adults. Applying fluocinolone acetonide here knocks down the inflammation so these spots fade back into the background.
Children with skin allergies sneak these itchy patches into everyday moments, making school or bedtime difficult. Pediatricians sometimes turn to milder strengths of fluocinolone acetonide, giving kids relief without raising risk for the thinning skin that stronger steroids cause.
Trust in fluocinolone acetonide comes from evidence and practical results. Research shows that short courses clear up symptoms better than basic moisturizers alone. Recapping facts from peer-reviewed journals: patients using medium-strength topical steroids for eczema achieve faster control and fewer sleepless nights. The U.S. Food and Drug Administration clears it for topical use, which means doctors and patients can rely on safety data stretching back decades.
As someone who’s experienced peeling hands in dry winters, I’ve seen how these creams make a difference in comfort at work and home. My dermatologist guided me through safe use—applying just a thin layer and never wrapping with plastic, which stops the drug from soaking in too deeply. Simple tips make these treatments safer.
People often think more medicine means faster results, but overusing topical steroids leads to real problems—skin thinning, stretch marks, even breaking down the skin’s natural barrier. Sticking with the right amount and following directions keeps those risks low. Whenever new or worsening infections pop up, it’s time to stop and ask a doctor for advice. Steroids can mask infection, letting things get worse without noticing.
Beyond prescriptions, regular moisturizing and avoiding harsh soaps guard against recurrences. Fluocinolone acetonide brings the worst symptoms under control, but good habits fill in the gaps between flare-ups. Many groups—from the American Academy of Dermatology to local patient advocates—offer resources for understanding treatment steps and spotting warning signs.
Using something like fluocinolone acetonide isn’t just a matter of following orders. Partnering with a doctor, tracking your skin’s ups and downs, and not getting complacent with overuse makes all the difference. Relief is within reach for most people willing to be both patient and proactive.
Fluocinolone Acetonide falls into that group of topical steroids doctors trust for eczema, psoriasis, and other skin rashes. Anyone who’s wrestled with relentless itching or inflamed patches knows relief matters more than fancy names. The key with these creams—especially potent ones like this—is about using just enough, just where it’s needed, and on guidance from a trusted healthcare professional.
Some folks think more cream speeds up results. In reality, piling it on raises the chances for side effects. Thinning skin, stretch marks, and flare-ups sneak up after days of overuse. For me, even a fingertip amount—often what dermatologists call the “fingertip unit”—tackles small rashes. Gently dabbing rather than rubbing it in keeps the medicine where the irritation lives, not all over healthy skin.
Anyone who’s had to balance daily life and rash relief has likely skipped a wash just to get relief sooner. Truth is, using Fluocinolone Acetonide on unclean or damp skin is less effective. Soap and water clear away oils and sweat that block the medicine. Patting dry, not scrubbing, avoids more redness. A few extra seconds can mean the difference between missing relief and getting it.
Doctors caution against using strong steroids like this around eyes, mouth, or broken skin. I’ve seen folks try to shrink puffiness or redness on eyelids, then get stuck with burning or thinning. These areas absorb the drug faster and raise risks. For those with eczema or psoriasis on the face, doctors sometimes switch to milder steroids or other creams to keep things safer.
Slathering moisturizers on top sounds harmless, but it can actually trap heat and drive the steroid deeper. Years back, I used to layer steroid cream and a thick emollient without thinking; ended up with stinging and more irritation. Spacing out applications—waiting 20 or 30 minutes between—is what most experts suggest so each product can do its job.
Even though packages come with instructions, every person’s skin has its own quirks. Some rashes want a thick ointment; others clear up with a light cream. For children or people with fragile skin, doses drop even lower. Getting all this straight from someone who knows your medical story saves time and prevents long-term harm.
Steroid creams like Fluocinolone Acetonide get a bad rap for a reason—overuse leaves its mark. If a rash spreads, oozes, or develops pain, calling the doctor makes more sense than adding extra cream. Skin isn’t just an outer covering; it tells the real story beneath. Short courses, small amounts, and return visits often work better than trying to invent home fixes.
Navigating chronic skin conditions goes past quick fixes. Using Fluocinolone Acetonide as instructed, pausing when told, and staying alert to side effects build trust with your doctor—and with your own skin. Relief comes not just from strong medicine, but from attention and respect for the body’s signals.
Fluocinolone acetonide, a corticosteroid used to address inflammation on the skin or inside the eye, can be a game changer for people dealing with eczema, psoriasis, or allergy-related rashes. The promise of calming itch and redness brings real hope during flares. Relief does not come without risk, though. Over years of working in community care and seeing people who use topical steroids, I have witnessed concern turn into frustration when unexpected effects show up.
At first, users tend to believe that a cream delivering so much comfort cannot cause trouble. Mild burning or stinging sometimes appears at the application site, especially right after starting treatment. Skin can dry out, thin, or develop stretch marks if someone uses the product for longer stretches than necessary. I have met patients whose skin bruised with only the lightest touch, after using steroids for months. These changes hit hardest in areas with thinner skin, like the face or underarms.
Hair growth in treated spots can pick up, and the skin sometimes lights up red or looks lighter than normal. These changes show up slowly, and more often among kids or those with sensitive skin. Sometimes, a rash comes back even harder when people stop the medicine too quickly, a rebound effect that sneaks up on those eager to quit.
Absorbing steroid through the skin does not usually lead to big changes elsewhere in the body. The risk of serious effects climbs when people use strong doses on large areas or under bandages, especially in children. Problems with hormone balance, like Cushing’s syndrome, have actually shown up in rare cases. Kids feel effects faster due to their thinner skin and faster metabolism.
Applied near the eyes, fluocinolone acetonide can trigger increased pressure inside the eye. This can set someone on a path toward glaucoma—not something that comes to mind when first picking up a cream or eye drop.
By calming the body’s natural inflammation, steroids slow down more than just redness and itch. The immune system’s reaction to bacteria and fungi gets weaker. This opens the door for infections, sometimes with odd-looking spots or pus where the cream or drops got used. A few people have shared stories of “unexplained” pimples or sores that kept coming back until a doctor took a closer look.
Whenever a doctor hands out a new prescription for fluocinolone acetonide, they need to give clear guidance on how much, where, and for how long. Long-term use or applying steroid to wide areas brings real risks, so I always encourage my patients to ask questions if they see thinning, bruising, or acne popping up in treated areas.
Checking your own skin for changes every few days helps. In my experience, those who keep in touch with their doctor and take breaks from treatment do much better in the long haul. Using less, taking short breaks, and choosing lower-strength creams for delicate skin all help avoid trouble down the line.
Listening to your skin—and getting regular check-ins with a pharmacist or doctor—makes a difference. It’s easy to focus on short-term comfort, especially during big flares, but paying attention to small changes in how skin or eyes feel often leads to better long-term outcomes. People do best when they keep the dialogue open, track side effects, and reach out early if something does not feel right. Side effects may sound scary, but with careful use and reliable advice, relief and safety can live together.
Fluocinolone acetonide works as a corticosteroid, easing itching, redness, and swelling. Over the years, many doctors have reached for it to calm flare-ups in eczema and other stubborn rashes. Talking with other parents, it's clear that skin problems become a real stress point—kids scratching at night, families looking for relief. Corticosteroids often step in during these rough patches.
Doctors regularly weigh risks and benefits before starting any medication in children. For fluocinolone acetonide, the evidence gives a mixed report card. The FDA lists it as safe for short-term use in children as young as three months old when used on the scalp and skin, so pediatricians do prescribe it for certain cases. Its gentle formulation, often in creams or shampoos, allows targeted treatment. Some studies back this up—short bursts for tough eczema can bring real relief, preventing infections from constant scratching.
Corticosteroid creams, including fluocinolone, have a reputation for thinning skin if used too hard or for too long. Children's skin drinks up medication faster, amplifying potential side effects. Thinning, bruising, and even problems with growth hormones stand out as risks. Researchers have pointed out that using the least strong steroid for the shortest time seems smart. Tracking symptoms and stopping once the skin recovers goes a long way.
Talking with pediatricians and parents, the approach feels practical: small tubes, careful instructions, and regular checkups. Parents often express worry about steroids, so real conversations matter. Many try gentler options first– unscented moisturizers, keeping fingernails trimmed, and a close eye on triggers like soaps or pollen. If nothing else works, fluocinolone sometimes comes out, cautiously and with a plan to taper it off.
Dermatologists count on measuring progress. Sometimes they use “fingertip units” to control dosing, avoiding over-application. From experience, teachers and daycare workers appreciate parents sharing if skin medication is in use—no one likes confusion if a rash appears at school.
Parents want to do the right thing, so education gives power. Doctors recommend using fluocinolone only on rough patches, not healthy skin, even if eczema keeps moving around. Taking breaks from steroid creams, known as “steroid holidays,” helps cut risks. Another tip is matching the cream to the body part—thicker skin like elbows handles steroids better than thin spots like eyelids.
Ointments and creams, though helpful, can’t solve everything. Allergy tests, better bathing habits, and mild laundry soaps all play a role. Newer, non-steroid creams such as tacrolimus give families another option when repeated flares frustrate everyone. Insurance coverage often decides what is realistic, causing another set of headaches for families and their doctors.
Watching a child suffer with eczema often breaks a parent’s heart. Still, short, careful use of fluocinolone under a doctor’s guidance can bring comfort. The key lies in respect: following instructions, knowing the risks, and checking in often. Staying alert to changes in a child’s skin lets families and doctors work together, aiming for healthy skin and happy days.
Fluocinolone acetonide gets prescribed for stubborn skin problems. This synthetic corticosteroid calms itch, quells redness, and can clear rashes that just won’t budge with over-the-counter creams. As someone who’s lived with persistent eczema, I’ve learned its strengths—and its hazards—up close.
The face, eyelids, groin, and armpits qualify as sensitive zones because skin is thin and absorbs medicines quickly. A dab of strong steroid like fluocinolone in these areas can soak in much deeper than on the palms or soles. I’ve watched friends use these creams for minor rashes under their eyes, only to wind up with thinning, paper-like skin after a couple of weeks.
Doctors speak up about this risk because thinning and stretch-mark-like scars can follow, especially on the face or in folds of the body. Fluocinolone acetonide belongs in the “medium to high potency” steroid family, which packs more punch than low-potency creams like hydrocortisone. The American Academy of Dermatology warns against strong topical steroids for more than a few days on delicate areas. Complications like rosacea, acne flare-ups, and skin color changes can linger long after the rash fades away.
Plenty of folks with stubborn skin diseases wrestle with the misery of flares that nothing else fixes. Dermatitis on the face or eyelids can keep people up at night. Over my years reporting on health, I’ve seen adults cry with relief when a steroid cream finally breaks the itch-scratch cycle. Still, the goal with fluocinolone acetonide should always be the shortest use possible—think days, not weeks. Dermatologists also often prescribe a weaker version or mix it with moisturizer to help stretch the benefits without the side effects.
Research supports that short-term, careful use—guided by an experienced dermatologist—brings the most improvement with the least trouble. Both the U.S. Food and Drug Administration and the National Eczema Association urge close follow-up, precise measurement of what gets applied, and a proper plan for stopping. For children or people with a past history of steroid reactions, all steroid use on the face or sensitive skin deserves double caution.
Non-steroid alternatives keep growing in popularity. Pimecrolimus and tacrolimus work differently from steroids and don’t carry the same thinning risk. Moisturizers, gentle cleansing, and allergy reduction keep many milder cases away from strong drugs in the first place. Stronger steroids save their power for the small sliver of cases that truly need it—ideally, after gentler choices get tried first.
After years navigating dermatology visits and my own steroid rashes, I see no quick fixes. Every patient brings a different story, but steroids like fluocinolone acetonide always demand respect, communication, and honest feedback with a doctor who cares. Doses, duration, and monitoring shape whether a short burst returns calm, or whether a moment of relief blurs into lifelong marks. The lesson: trust real experience, trust science, and never treat the face like just another patch of skin.
| Names | |
| Preferred IUPAC name | (6α,11β,16α)-6,9-Difluoro-11,21-dihydroxy-16,17-[(1-methylethylidene)bis(oxy)]pregna-1,4-diene-3,20-dione |
| Other names |
Fluocinolone
Fluocinolone acetonidum Flucort Synalar Derma-Smoothe/FS Capex Retisert |
| Pronunciation | /fluːˌoʊ.sɪˈnoʊ.loʊn əˈsiː.təˌnaɪd/ |
| Preferred IUPAC name | (6α,11β,16α)-6,9-Difluoro-11,21-dihydroxy-16,17-[(1-methylethylidene)bis(oxy)]pregna-1,4-diene-3,20-dione |
| Other names |
Fluorocinolone acetonide
Fluocinolone diacetate Synalar Fluonid Flucort Lidex Retisert |
| Pronunciation | /fluːˌoʊ.sɪˈnəʊ.ləʊn ˌæs.ɪˈtɒ.nɪd/ |
| Identifiers | |
| CAS Number | 3424-82-6 |
| Beilstein Reference | 3921105 |
| ChEBI | CHEBI:31422 |
| ChEMBL | CHEMBL1202 |
| ChemSpider | 21476487 |
| DrugBank | DB00180 |
| ECHA InfoCard | 100440 |
| EC Number | 5.3.1.9 |
| Gmelin Reference | 87812 |
| KEGG | C14430 |
| MeSH | D005473 |
| PubChem CID | 5745 |
| RTECS number | GL9575000 |
| UNII | UP5R295LS1 |
| UN number | UN number not assigned |
| CompTox Dashboard (EPA) | DTXSID8044525 |
| CAS Number | 3424-82-6 |
| Beilstein Reference | 1302202 |
| ChEBI | CHEBI:4493 |
| ChEMBL | CHEMBL1202 |
| ChemSpider | 207346 |
| DrugBank | DB00180 |
| ECHA InfoCard | 02-212-002013 |
| EC Number | EC 3.1.3.16 |
| Gmelin Reference | 69236 |
| KEGG | C14412 |
| MeSH | D005473 |
| PubChem CID | 5281064 |
| RTECS number | MU1400000 |
| UNII | BKJ8M8G8Y8 |
| UN number | Not assigned |
| CompTox Dashboard (EPA) | DTXSID9021399 |
| Properties | |
| Chemical formula | C24H30F2O6 |
| Molar mass | 452.49 g/mol |
| Appearance | White to almost white crystalline powder |
| Odor | Odorless |
| Density | 1.31 g/cm³ |
| Solubility in water | Insoluble in water |
| log P | 2.56 |
| Acidity (pKa) | 12.53 |
| Basicity (pKb) | 12.42 |
| Refractive index (nD) | 1.627 |
| Viscosity | Viscous liquid |
| Dipole moment | 2.28 D |
| Chemical formula | C24H30F2O6 |
| Molar mass | 452.496 g/mol |
| Appearance | White to almost white crystalline powder |
| Odor | Odorless |
| Density | 1.31 g/cm³ |
| Solubility in water | Slightly soluble in water |
| log P | 2.56 |
| Vapor pressure | 2.8 × 10⁻⁹ mmHg |
| Acidity (pKa) | 12.36 |
| Basicity (pKb) | 13.79 |
| Magnetic susceptibility (χ) | -8.8e-6 |
| Refractive index (nD) | 1.607 |
| Viscosity | Viscous liquid |
| Dipole moment | 2.36 D |
| Pharmacology | |
| ATC code | D07AC04 |
| ATC code | D07AB02 |
| Hazards | |
| Main hazards | May cause eye, skin, and respiratory tract irritation. |
| GHS labelling | GHS labelling: Signal word: Warning; Hazard statements: H315, H319, H335; Pictograms: GHS07 (Exclamation mark) |
| Pictograms | GHS07, GHS08 |
| Signal word | Warning |
| Hazard statements | May cause an allergic skin reaction. Causes serious eye irritation. |
| Precautionary statements | Keep out of reach of children. For external use only. Avoid contact with eyes. If irritation develops, discontinue use and consult a physician. Do not use on broken or infected skin unless directed by a doctor. |
| NFPA 704 (fire diamond) | Health: 2, Flammability: 1, Instability: 0, Special: - |
| Flash point | > 291.8 °C |
| Lethal dose or concentration | LD50 (Rat, oral): >3,000 mg/kg |
| LD50 (median dose) | LD50 (median dose) of Fluocinolone Acetonide: "1g/kg (oral, rat) |
| NIOSH | MF9275000 |
| PEL (Permissible) | Not established |
| REL (Recommended) | 10 mg |
| Main hazards | May cause irritation to skin, eyes, and respiratory tract; prolonged exposure may cause allergic reactions or systemic effects. |
| GHS labelling | GHS07 |
| Pictograms | Eye Irritant, Skin Sensitizer, Health Hazard |
| Signal word | Warning |
| Hazard statements | Hazard statements": "Not a hazardous substance or mixture according to the Globally Harmonized System (GHS). |
| Precautionary statements | Keep out of reach of children. For external use only. Avoid contact with eyes. Use only as directed by a healthcare professional. Do not use on broken or infected skin unless directed. Discontinue use if irritation or sensitivity occurs. |
| Flash point | > 260.6 ± 27.7 °C |
| Lethal dose or concentration | LD50 (Rat, oral): >3000 mg/kg |
| LD50 (median dose) | LD50 (median dose): Oral (rat): >3000 mg/kg |
| NIOSH | MN9285000 |
| PEL (Permissible) | Not established |
| REL (Recommended) | 0.01% |
| Related compounds | |
| Related compounds |
Cinolone acetonide
Triamcinolone acetonide Fluocinolone Fluocinonide Dexamethasone Prednisolone Hydrocortisone Betamethasone |
| Related compounds |
Triamcinolone acetonide
Fluocinolone Fluorometholone Dexamethasone Betamethasone |
| Thermochemistry | |
| Std enthalpy of formation (ΔfH⦵298) | -1096.7 kJ/mol |
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