Prednisone got its start in the 1950s, at a time when medicine scrambled for answers against inflammation and immune disorders. Arthur Nobile managed to pull off the chemical transformation that set Prednisone apart during his work at Schering Corporation. Before that, doctors relied on cortisone, but the body had to do some heavy lifting to convert it into something active. Prednisone, though, needed only the liver for activation, making it more predictable and fitting for oral use. This chemical leap meant safer, more reliable arthritis and asthma control for millions. Over the years, its spot in treatment plans grew as research pulled together more evidence showing it could control a staggering range of immune-related problems, from lupus to organ rejection.
Prednisone is a synthetic corticosteroid that steps in to tamp down inflammation and suppress immune responses. The pill form gives it an edge for outpatient care, letting people manage their diseases from home. For conditions such as severe allergies, rheumatoid arthritis, asthma flare-ups, and even certain cancers, Prednisone helps turn the tide on swelling, pain, and immune overactivity. Doctors pick it because it gets into the bloodstream quickly and reliably, reaching almost every cell. Years of use have kept it part of emergency kits and long-term management for many patients. Unlike some newer treatments, Prednisone’s general affordability ensures access isn’t limited to just those with premium insurance or deep pockets.
Prednisone shows up as a white, odorless crystalline powder. It doesn’t dissolve all that well in water, but it mixes better with alcohol and some organic solvents. Its melting point hovers between 220°C and 230°C. The molecule’s structure belongs to the corticosteroid family, built around a cyclopentanoperhydrophenanthrene skeleton, with 17 carbon atoms and distinct double bonds that guide its activity in the human body. Molecular formula C21H26O5, its pharmacokinetics vary with liver health, which can change the dose needed for the same result. Stability under routine storage conditions lets pharmacies stock it in bulk, and it keeps well when kept from excessive heat and light.
Prednisone tablets typically contain anywhere from 1 mg to 50 mg per dose. Each tablet’s label spells out the strength, the manufacturer, the batch number for tracking, as well as government approval codes. U.S. Pharmacopeia standards demand that every batch sits within tightly defined purity limits, with impurities capped below regulatory thresholds. Packages display warnings about taking it with food, avoiding sudden withdrawal, and keeping it out of children’s reach. Information about expiry and storage conditions is front and center. For hospital use, injectable forms include the compound dissolved in sterile liquid with preservatives, and labels carry special handling and emergency antidote instructions.
Industrial-scale production of Prednisone starts with plant sterols, usually diosgenin drawn from yams or soybeans. Chemists run a series of oxidation reactions to introduce the right oxygen atoms into the backbone, then tune the double bonds through selective hydrogenation or dehydrogenation. In my experience working alongside chemical engineers, these steps need constant temperature checks and precision in handling reagents like chromium trioxide or selenium dioxide—errors here can leave you with a useless side product. The final compound runs through repeated purification, including recrystallization, charcoal adsorption, or chromatography, until the drug meets pharmaceutical purity grades. This rough-and-tumble process brings the raw steroid closer to a medicine people and hospitals can rely on.
Prednisone acts as a prodrug, so once swallowed, liver enzymes—specifically 11β-hydroxysteroid dehydrogenase—convert it to prednisolone, the compound that does the work on tissues. Modifications of prednisone itself generally target its activation speed or how long it stays in the body. Chemical tweaks can add or remove functional groups, or insert protective groups to shield it from stomach acid, resulting in derivatives used for more specialized conditions. Synthetic chemists sometimes attach acetate or phosphate groups for improved solubility in injectable forms, letting it act faster in emergencies. Each alteration brings its own differences in how the drug is absorbed, how far it travels in the body, or how strong its effects are on immune cells.
Prednisone goes by more than one name in pharmacies and hospitals worldwide. Some of these are trade names from major producers: Deltasone, Rayos, and Sterapred are common ones in the United States. Generic options remain widely available, reducing costs and improving accessibility. Synonyms include 17,21-dihydroxypregna-1,4-diene-3,11,20-trione or dehydrocortisone for chemical records, but nobody except regulatory agencies or the manufacturing plant needs to know those in regular conversation. Doctors and patients most often stick with Prednisone, given its long and steady history of use.
Prednisone requires clear procedure in healthcare settings. Overuse runs the risk of side effects—weight gain, brittle bones, mood changes, and blood sugar spikes count among the most common. Drug information leaflets warn people not to stop suddenly, as the adrenal glands need time to restart their own steroid production. Hospitals follow strict protocols for handling and administering high doses, double-checking patient records to prevent overdose or conflicts with other medicines. Personal protective equipment helps pharmacy workers avoid accidental exposure during manufacturing and preparation. On the regulation side, agencies like the FDA and EMA audit plants and check quality records regularly, to catch any drift from safety or quality standards.
In everyday medical practice, doctors rely on Prednisone for conditions that involve out-of-control inflammation. This includes severe asthma, autoimmune diseases like lupus, allergies that threaten airway closure, and flares of inflammatory bowel disease. After a kidney transplant or to manage immune complications in cancer therapy, Prednisone steps in to hold the body’s own defense systems in check, reducing the risk of organ rejection or collateral immune damage. Dermatologists use it for sudden flares in conditions like eczema or severe poison ivy. Sometimes, short-term high doses—known as pulse therapy—help patients escape dangerous inflammatory episodes. Guidelines keep shifting as researchers test how short or long these treatments should last, always balancing benefit against side effect risks.
Research into Prednisone’s effects and side effects never really stops. Investigators continue to explore how genetic differences affect metabolism, which can change who gets benefit versus risk of long-term problems. Some clinical trials look at pairing Prednisone with other immunosuppressants, to try to cut doses and shrink the side effect burden. Pharmacologists experiment with controlled-release tablets that release Prednisone at specific times of day—linked to the body’s natural hormone cycles—to improve results in rheumatoid arthritis and asthma. Scientists also study how to target the drug more precisely to inflamed tissues, reducing exposure elsewhere. Ongoing data come from patient registries, digital health tools, and newer imaging techniques, expanding the foundation for better dosing and monitoring strategies.
Toxicity is not just theory with Prednisone. Investigators track short-term and chronic effects, ranging from blood pressure elevation and fluid retention to risk of psychosis and osteoporosis. Animal studies shaped early knowledge—high doses cause stunted growth, muscle weakness, and adrenal gland atrophy in rats and dogs—patterns echoed in humans on long courses. Population data from decades of use show diabetes, easy bruising, and cataracts in people who stay on Prednisone for over a few weeks. Recent research picks apart which side effects come from dose size, which from treatment length, and which patients face the highest risks. Physicians draw on this evidence every day, working to balance relief from illness with the reality of potential harm.
Prednisone’s future looks tied to both its strengths and its limits. Newer drugs may eclipse it in some specialized diseases, yet cost and reliability keep Prednisone within reach in both high- and low-resource settings. Researchers continue to search for chemical tweaks that cut down on metabolic and bone side effects while preserving anti-inflammatory power. Digital health tools promise better monitoring, catching trouble early and letting doctors adjust doses faster. Some teams work toward rapid saliva or blood tests to spot dangerous blood sugar swings or adrenal suppression before things get out of hand. As immune science grows more precise, personalized dosing may step in, tailoring use and limiting risk like never before. For now, Prednisone remains the workhorse corticosteroid for countless patients, with new generations of researchers driving efforts to boost its benefit and limit its toll.
Doctors give prednisone to people battling inflammation. It acts fast, often showing results in a couple of days. I’ve seen friends with asthma who could barely breathe get up, move, and talk freely after starting a course. This drug tampers down the body's immune system, which sounds strange since we’re told we need our immune system strong. Yet, overactivity causes damage in some diseases like lupus, rheumatoid arthritis, and severe allergies. Prednisone dials that reaction back.
Some diseases throw the immune system into overdrive. My neighbor, fresh out of surgery, got thrown onto prednisone after her joints swelled beyond understanding. The pill didn’t just take down the inflammation; it kept her body from attacking itself. In cases of sudden asthma flares or allergic reactions, a quick course can mean the difference between an ER trip and a regular day.
Children and adults with conditions like Crohn’s disease, ulcerative colitis, and multiple sclerosis also get prescribed steroid bursts. Skin rashes—like poison ivy or eczema—sometimes call for a dose too. Prednisone earns its keep in hospitals and clinics for this broad usefulness.
Long-term use of prednisone comes with a price tag you can’t ignore. Weight gain, high blood sugar, brittle bones, higher risk of infection—it’s a long list. I’ve watched someone struggle through mood swings, sleeplessness, and even anxiety while on just a few weeks of this pill. Doctors monitor patients closely, checking bone density, blood sugar, and infection markers.
Coupled with prednisone’s ability to save lives, there’s the lurking risk of addiction—not to the drug itself but to the relief it brings. People find it tough to come off prednisone when pain flares or autoimmune symptoms return. Quitting suddenly is dangerous, because the body’s own steroid production slows down; tapering down over weeks, sometimes months, is the only route.
Many people have no idea they should adjust their diet and stay active while on steroids. Doctors advise boosting calcium intake, cutting out extra salt, and checking blood pressure. People who don’t watch these can find themselves with high blood pressure or weaker bones. Using the lowest effective dose, for the shortest time possible, helps lower risk.
Some researchers search for safer alternatives. New medications called “steroid-sparing agents” or targeted biologics come with their own sets of risks and benefits. These options can take weeks or months to kick in, though, making prednisone a stop-gap for severe attacks.
Patients must walk hand in hand with their doctors on prednisone. I’ve seen lives transformed, both for better and worse, because of this drug. Open conversation and monitoring mean fewer surprises. Reading up, questioning, getting regular check-ups, and watching for side effects helps anyone stuck with a prednisone prescription. Medicine works best when people taking it know what’s at stake and stay part of the decision-making process.
Prednisone often gets prescribed for everything from asthma and serious allergies to arthritis and stubborn skin rashes. When doctors pull this medication out, it usually means your body needs some strong help cooling down inflammation. But just about everyone who’s been on prednisone for more than a few days has probably wrestled with the drawbacks that can tag along.
Start taking prednisone, and you may notice your appetite ramp up. This change doesn’t just mean you grab an extra snack now and then—many people gain extra pounds over a few weeks. That hunger boost also pairs with extra fluid stuck in tissues, which leaves the face looking puffier than normal. A lot of folks call it the “moon face.” Press your socks for a second and you might see the swelling linger around your ankles.
Stomach lining takes a beating, too. Acid production climbs, so people get heartburn or even ulcers, especially if coffee or spicy meals already trigger symptoms. A glass of milk and food with every pill lessens this, but doesn’t erase the risk. Insomnia shows up for some within the first week, and every extra day on prednisone seems to nudge along trouble staying asleep. Over time, bones tend to become brittle, which means the longer someone takes steroids, the higher the chance of fractures.
Nothing quite prepares you for the emotional swings that can roll out. Mood shifts for some get intense—tears during a sitcom, a short fuse in traffic, even racing thoughts at bedtime. Someone in my family kept a journal when prednisone hit double digits for a tough illness. Looking back, we could chart crankiness and sudden worries right alongside increases in dosage. It felt easier to track reality versus the drug’s mental whiplash that way.
Science backs this up. Data from the National Institutes of Health suggests almost two thirds of people on long-term prednisone feel anxious or struggle with mood swings. Friends and family may think it’s just stress from being sick, but those chemical shifts are real. That makes honest conversation and support at home critical.
Prednisone tamps the immune system down. With a lower defense line, colds and infections hit harder. My neighbor, a retired nurse, always told me—avoid the crowds, use sanitizer, and skip uncooked foods during these stretches. Active monitoring by blood tests can tell doctors when it’s safe to let up.
Patients shouldn’t shoulder these challenges alone. Nutritionists can build menus with extra calcium, lower sodium, and controlled calories to stave off weight and bone loss. Pharmacists can suggest medication timing to help handle insomnia. Honest feedback to your doctor about how you’re feeling—physically and emotionally—sometimes leads to a lower dose or slower taper down the line.
Prednisone changes routines. The side effects aren’t always minor or fleeting, and they shape daily life. Giving people space to talk about those shifts—without judgment—lets them deal with what’s hard, not just the illness itself. That’s a lesson learned through many kitchen table conversations at home, and it still rings true.
Prednisone treats dozens of conditions, from asthma flare-ups to stubborn rashes and even autoimmune troubles. This medication doesn’t work like a simple painkiller. It’s a steroid, and steroids can do a lot more than just cool off an angry joint or help a cough fade away. But with power comes the possibility of headaches—sometimes quite literally.
Doctors hand out careful instructions for a reason. I remember my own handful of days on Prednisone for a bad case of poison ivy. My doctor stressed, “Don’t stop suddenly. Don’t change the dose unless I say so.” This isn’t just old-school caution. The body’s adrenal glands count on us to take steroids the right way. Prednisone acts much like our own cortisol; when the body gets its daily dose from a pill, it takes a break from making its own.
Some folks need to take their dose all at once in the morning, others divide it across the day. There’s a reason mornings often win: the body’s natural rhythm pumps out most cortisol around breakfast time. Taking the pill in sync with that schedule helps keep things steady and cuts down on sleepless nights or jittery afternoons.
Missed a dose? The best advice comes down to checking the clock. If it’s only been a few hours, take it as soon as possible. If it’s almost time for the next dose, don’t double up. Skipping or doubling doses can mess with blood sugar, mood, and blood pressure.
Prednisone can save lives. But steroids come with a long list of possible side effects—weight gain, mood swings, high blood pressure, higher risk of infection. From personal experience and years of health writing, I’ve heard plenty of regrets about not taking weak bones, diabetes, or ulcers seriously during longer courses. The higher the dose and the longer the treatment, the more attention those risks deserve.
The immediate side effects also pack a punch. Heartburn, muscle weakness, trouble sleeping, and a face that seems to puff up overnight. Regular checkups help spot these problems before they spiral out of control.
Sudden stops can be dangerous because the adrenal glands won’t jump to attention fast enough. Tapering works, and only a doctor can know the right speed. Some people need months to come off Prednisone after a long run. Others might just need a week or two of gently decreasing doses. Withdrawal can bring headaches, fatigue, body aches, or depression.
Food can cushion the blow to the stomach, so most doctors advise swallowing Prednisone after a meal or with a snack. Give up on alcohol and limit salt and sugar for these weeks or months. Bone and muscle strength take a hit, so getting some sunlight, lifting a few weights, or walking every day pulls double duty.
A medication list and a phone number for your clinic matter, too. Even vaccines change with Prednisone in the picture—some shots shouldn’t be given until you’re off. Prednisone deserves respect. Listening to your prescriber, staying aware of side effects, and keeping track of every pill make the journey safer and easier.
Prednisone sits in a unique corner of medicine. The first time a doctor handed me a prednisone script, I thought of it almost like a strong painkiller. I only learned better after watching my family member go through a swollen knee episode. The doctor handed over the little white pills and explained the benefit, but she spent more time on the risks—mostly, the danger of dropping prednisone cold turkey.
Our bodies make a hormone called cortisol through the adrenal glands. Prednisone, a type of corticosteroid, mimics cortisol. With regular prednisone use, the body thinks, “Hey, I don’t need to make as much of my own stuff.” The longer someone takes this drug—and the higher the dose—the lazier those adrenal glands become. I’ve seen a friend get prescribed prednisone for weeks for his Crohn’s flare. The plan always included a taper: a carefully timed reduction in dose. Doctors don’t enjoy complicating the schedule unless it’s necessary. The taper isn’t about bureaucracy—it’s about survival.
Stopping suddenly can bring on adrenal insufficiency. Fatigue turns crushing, the stomach churns, the body feels harder than it should. Unchecked, the danger spirals into a real emergency: blood pressure drops, and the heart can’t pump the way it should. I remember a guy who thought the rules didn’t apply to him. He said, “Let’s just get it over with.” He got lucky. His shaking, nausea, headaches landed him in the hospital with adrenal crisis—but he pulled through. Not everyone does.
Ask anyone who’s taken prednisone for a month or more: it doesn’t just chase away swelling or pain. It changes hunger. It puffs up faces. Bones can thin out. Prednisone raises blood sugar. For a diabetic, quitting suddenly means more than lost energy; blood glucose can swing out of control. I’ve seen family members rage, cry, or sleep all day during a steroid taper. Sometimes, people chalk it up to their old illness coming back, but withdrawal can bring its own storm.
Doctors know the withdrawal problem well. They want to keep people safe, so they tailor tapers to each person’s dose, reason for the drug, and time spent on it. Roughly speaking, a week or less on prednisone lets people off the hook; beyond that, sudden stops spell danger.
Trust in this area remains everything. If the side effects become rough, the urge to quit grows. Some folks get frustrated when their skin breaks out or sleep falls apart. Turning to online groups, I’ve seen patients encourage each other to call the clinic, not just skip pills. This pointed me to the solution that works: team up with the prescriber, no matter how long—or short—the steroid course runs.
Prednisone serves a purpose, from asthma attacks to stubborn rashes or autoimmune flares. But the body gets used to it fast. Doctors, pharmacists, and patients can help by keeping communication honest and open. The safest path weaves between listening to medical advice and reporting every new symptom. Every dose comes with a plan, a goal, and a way out—never an off switch.
Expecting a baby changes the way people see every pill bottle in the house. Doctors hand out prescriptions for many reasons, and prednisone often comes up because it brings powerful relief for asthma, allergies, lupus, or other autoimmune struggles. I remember my own relative facing flares in pregnancy, clutching her inhaler and prescription sheet, wondering if prednisone would calm things down without causing trouble for her unborn baby.
Scientists know prednisone crosses the placenta in small doses, just enough to reach the baby. For certain conditions, this medicine becomes a necessity. Uncontrolled illness in a pregnant person damages both mother and fetus more than cautious, supervised steroid use. Studies back this up: the American College of Rheumatology notes that, although long-term high doses have been linked to low birth weight or cleft palate in rare cases, short bursts or lower daily doses don’t bring the same level of concern. Most babies are born healthy.
The web overflows with rumors—every search turns up a horror story or a message board full of half-truths. But let’s talk about facts. Researchers reviewed thousands of cases: short-term prednisone use hasn’t increased miscarriage rates. A handful of studies suggest tiny changes in birth outcomes, but not enough to cancel treatment where it’s needed.
Doctors do keep a close eye on dose and duration. If a person can manage their condition with as little steroid as possible, they do. Sometimes switching to inhalers or topical steroids makes sense, because these options send less medicine throughout the body.
New parents worry their medicine may slip into breast milk. Here’s what evidence shows: small amounts of prednisone do pass into milk, but not in levels high enough to threaten a nursing infant. The American Academy of Pediatrics calls prednisone “compatible” with breastfeeding. They recommend waiting a few hours after a dose if taking more than 20 mg daily, letting the level in milk drop even more before breastfeeding again.
Most lactating people on prednisone keep nursing. Pediatricians rarely see problems. It feels reassuring as someone who has seen close friends juggle medication and newborn care without giving up either.
Nobody should rely on anonymous online hearsay or expect a doctor to rubber-stamp every request. A board-certified obstetrician takes into account the full story: stage of pregnancy, dose, underlying disease, and family wishes. Sometimes patients need more reassurance. Trusted sources like the Mayo Clinic and the National Institutes of Health offer clear breakdowns on prednisone’s real risks, matching the guidance people get face-to-face at a prenatal visit.
Families juggling chronic illness and pregnancy deserve respect, not judgment. Communication between patient, doctor, and pharmacist remains the best tool. People might feel a shock to discover prednisone on their list, but with careful monitoring, most pregnancies continue safely.
Medical research never stops looking for safer, more targeted ways to manage disease in pregnancy and lactation. Until then, community support and accurate information matter most. Honest conversations plus solid science go further than fear.
| Names | |
| Preferred IUPAC name | 17,21-dihydroxypregna-1,4-diene-3,11,20-trione |
| Other names |
Deltasone
Rayos Sterapred |
| Pronunciation | /ˈprɛd.nɪˌsoʊn/ |
| Preferred IUPAC name | 17,21-Dihydroxypregna-1,4-diene-3,11,20-trione |
| Other names |
Deltasone
Rayos Prednicot Sterapred Sterapred DS Winpred |
| Pronunciation | /ˈprɛd.nɪ.soʊn/ |
| Identifiers | |
| CAS Number | 53-03-2 |
| 3D model (JSmol) | `3D model (JSmol)` string for Prednisone: ``` CC(=O)[C@H]1CC[C@H]2[C@@H]3CCC4=CC(=O)C=C[C@@]4([C@]3([C@H](C[C@@]12C)O)C)C ``` |
| Beilstein Reference | 1520263 |
| ChEBI | CHEBI:5746 |
| ChEMBL | CHEMBL1440 |
| ChemSpider | 54680682 |
| DrugBank | DB00635 |
| ECHA InfoCard | 06e1bc7a-3c50-4e0a-bc3a-431f09b8137c |
| EC Number | 2.3.1.50 |
| Gmelin Reference | 70877 |
| KEGG | C00247 |
| MeSH | D004364 |
| PubChem CID | 5755 |
| RTECS number | **RY0900000** |
| UNII | XBX3Y70FTX |
| UN number | UN number not assigned |
| CAS Number | 53-03-2 |
| 3D model (JSmol) | `3Dmol.js?query=Prednisone` |
| Beilstein Reference | 3580733 |
| ChEBI | CHEBI:8637 |
| ChEMBL | CHEMBL645 |
| ChemSpider | 5461 |
| DrugBank | DB00635 |
| ECHA InfoCard | 100.042.409 |
| EC Number | 3.2.1.33 |
| Gmelin Reference | 17323 |
| KEGG | D00477 |
| MeSH | D011238 |
| PubChem CID | 5755 |
| RTECS number | UD2060000 |
| UNII | PYL8F0PV7J |
| UN number | UN2811 |
| CompTox Dashboard (EPA) | urn:uuid:50e5a43b-f6db-4e55-bc53-9964978b7b7a |
| Properties | |
| Chemical formula | C21H26O5 |
| Molar mass | 358.43 g/mol |
| Appearance | Prednisone appears as a white to off-white, odorless, crystalline powder. |
| Odor | Odorless |
| Density | 1.27 g/cm³ |
| Solubility in water | Slightly soluble in water |
| log P | 1.56 |
| Vapor pressure | Vapor pressure: 4.7E-12 mmHg |
| Acidity (pKa) | 12.3 |
| Basicity (pKb) | 12.42 |
| Magnetic susceptibility (χ) | -10.7×10⁻⁶ cm³/mol |
| Refractive index (nD) | 1.589 |
| Dipole moment | Prednisone dipole moment: "2.56 D |
| Chemical formula | C21H26O5 |
| Molar mass | 358.43 g/mol |
| Odor | Odorless |
| Density | 1.33 g/cm³ |
| Solubility in water | Practically insoluble in water |
| log P | 1.56 |
| Vapor pressure | Prednisone does not have a measurable vapor pressure (practically non-volatile). |
| Acidity (pKa) | 12.38 |
| Basicity (pKb) | pKb = 11.06 |
| Refractive index (nD) | 1.593 |
| Dipole moment | Prednisone dipole moment: "4.58 D |
| Thermochemistry | |
| Std molar entropy (S⦵298) | 316.8 J·mol⁻¹·K⁻¹ |
| Std enthalpy of formation (ΔfH⦵298) | -686.2 kJ/mol |
| Std enthalpy of combustion (ΔcH⦵298) | -6504 kJ/mol |
| Std molar entropy (S⦵298) | 416.4 J·mol⁻¹·K⁻¹ |
| Std enthalpy of formation (ΔfH⦵298) | -789.7 kJ/mol |
| Std enthalpy of combustion (ΔcH⦵298) | -6434 kJ/mol |
| Pharmacology | |
| ATC code | H02AB07 |
| ATC code | H02AB07 |
| Hazards | |
| Main hazards | May cause immunosuppression, increased risk of infections, hyperglycemia, osteoporosis, mood changes, hypertension, gastrointestinal irritation, and adrenal suppression. |
| GHS labelling | GHS05, GHS07 |
| Pictograms | `"POM, L01, N01, D01, A01"` |
| Signal word | Warning |
| Hazard statements | No hazard statements. |
| Precautionary statements | Keep out of reach of children. Use only as directed by your doctor. Do not share this medication with others. Store at room temperature away from moisture and heat. |
| Autoignition temperature | 360 °C |
| Lethal dose or concentration | LD50 (oral, rat): 1,500 mg/kg |
| LD50 (median dose) | 4720 mg/kg (rat, oral) |
| NIOSH | RXCUI:8630 |
| PEL (Permissible) | PEL (Permissible Exposure Limit) for Prednisone: Not established |
| REL (Recommended) | Prednisone: 50 mg |
| IDLH (Immediate danger) | Not Established |
| Main hazards | May cause immunosuppression, increased risk of infection, adrenal suppression, osteoporosis, hyperglycemia, hypertension, mood changes, peptic ulcers, and delayed wound healing. |
| GHS labelling | GHS labelling of Prednisone: `"Hazard statements: H361 - Suspected of damaging fertility or the unborn child. Precautionary statements: P201, P202, P280, P308+P313, P405, P501. Signal word: Warning. Pictogram: Health hazard."` |
| Pictograms | breastfeeding,no-driving,pregnancy-alert,warning-4,alcohol-interaction,food-interaction,rx-only |
| Signal word | Warning |
| Hazard statements | No hazard statements. |
| Precautionary statements | Keep out of reach of children. Use only as directed by your doctor. Do not share with others. Store in a cool, dry place away from light. Dispose of properly if no longer needed. |
| NFPA 704 (fire diamond) | 1-0-0 |
| Lethal dose or concentration | LD50 (oral, rat): >3000 mg/kg |
| LD50 (median dose) | LD50 (median dose) of Prednisone: 1,500 mg/kg (oral, rat) |
| NIOSH | NIOSH: DD4556000 |
| PEL (Permissible) | Not established |
| REL (Recommended) | 5-60 mg/day |
| Related compounds | |
| Related compounds |
Prednisolone
Hydrocortisone Methylprednisolone Dexamethasone Triamcinolone |
| Related compounds |
Prednisolone
Hydrocortisone Cortisone Dexamethasone Methylprednisolone Triamcinolone |
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